What is HCG and how does it function as an LH analog?

HCG (Human Chorionic Gonadotropin) is a naturally occurring glycoprotein hormone composed of an α-subunit (shared with LH, FSH, and TSH) and a unique β-subunit (hCGβ) determining hormone-specific biological activity. HCG acts as a functional LH (luteinizing hormone) analog - binding to the LH/CG receptor (LHCGR, a Gs-coupled GPCR) on Leydig cells (testes) and theca/luteal cells (ovaries) with high affinity, triggering identical downstream signaling cascades as native LH. Key functions include: (1) Steroidogenesis stimulation - HCG activates LHCGR → Gs-coupled cAMP-PKA signaling → upregulates StAR protein (facilitates cholesterol transport into mitochondria) → activates P450scc and other steroidogenic enzymes → increases testosterone production (males) or progesterone/estrogen (females). (2) Gonadal support - maintains Leydig cell testosterone secretion and luteal progesterone production. (3) Extended half-life - HCG's heavily glycosylated β-subunit provides ~24-36 hour half-life vs LH's ~20-30 minutes, enabling sustained LHCGR activation and prolonged steroidogenic effects. (4) Pregnancy maintenance - physiologically, HCG is secreted by placental syncytiotrophoblasts to maintain corpus luteum progesterone production preventing menstruation. Research applications include LHCGR pharmacology studies, steroidogenesis pathway investigations, gonadotropin signaling research, fertility and reproductive physiology, testosterone regulation mechanisms, comparative gonadotropin studies (HCG vs LH vs FSH), pharmaceutical development of fertility therapeutics, and testicular/ovarian cell biology. HCG's LH-mimetic properties combined with extended pharmacokinetics make it valuable for investigating sustained gonadotropin effects vs pulsatile native LH, LHCGR desensitization mechanisms, and chronic steroidogenic stimulation in laboratory models.

How does HCG compare to native LH in receptor binding and steroidogenesis?

HCG and LH share the same receptor (LHCGR) and activate identical signaling pathways, but differ in pharmacokinetics and clinical/research applications: (1) Receptor binding - both bind LHCGR with high affinity (low nanomolar Kd), HCG may show slightly higher affinity due to additional glycosylation-mediated receptor interactions; both trigger Gs-coupled cAMP-PKA activation. (2) Half-life - HCG: ~24-36 hours (extensive β-subunit glycosylation protects from renal clearance); native LH: ~20-30 minutes (rapid degradation and clearance) - this >40-fold difference fundamentally changes pharmacodynamics. (3) Steroidogenic potency - both maximally stimulate testosterone/progesterone production via identical mechanisms (cAMP → PKA → StAR → steroidogenic enzymes), but HCG's extended half-life provides sustained elevation vs LH's pulsatile effects. (4) Physiological patterns - native LH secreted in pulsatile bursts (hypothalamic GnRH-driven) critical for normal gonadal function; HCG provides non-pulsatile sustained stimulation mimicking pregnancy physiology. (5) Research applications - native LH ideal for studying physiological pulsatile gonadotropin effects, GnRH-LH axis regulation, and acute LHCGR activation; HCG preferred for sustained steroidogenesis research, chronic LHCGR stimulation studies, receptor desensitization mechanisms, and pharmaceutical modeling (longer-acting). (6) Dosing - HCG's extended half-life enables less frequent dosing (every 2-3 days) vs LH requiring continuous or very frequent administration for sustained effects. (7) Clinical translation - HCG is pharmaceutical standard for fertility treatments due to stability and half-life; recombinant LH available but less commonly used. (8) Receptor desensitization - chronic HCG exposure downregulates LHCGR expression and uncouples receptor from signaling (desensitization), valuable for studying gonadotropin tolerance mechanisms. Choose native LH for physiological pulsatility studies; choose HCG for sustained steroidogenesis, chronic LHCGR activation, and pharmaceutical research.

What purity and quality standards does your HCG meet?

Our HCG exceeds >98% purity verified by HPLC with comprehensive pharmaceutical-grade quality control for this complex glycoprotein hormone. Each batch undergoes: reverse-phase HPLC demonstrating >98% purity with proper retention time for intact HCG heterodimer (α+β subunits), size-exclusion chromatography (SEC) confirming correct molecular weight (~37-39 kDa depending on glycosylation extent - significantly larger than peptide hormones due to carbohydrate moieties), mass spectrometry molecular weight validation and subunit identification (α-subunit ~14.5 kDa, β-subunit ~22-24 kDa with extensive N- and O-linked glycosylation), glycosylation analysis verifying proper carbohydrate attachment (critical for bioactivity and half-life - HCGβ has 4 N-glycosylation sites and 4 O-glycosylation sites), intact heterodimer verification confirming non-covalent α-β association (active form; dissociated subunits inactive), LHCGR binding bioassays measuring HCG's affinity for LH/CG receptor using receptor competition assays or cell-based binding studies (Kd typically low nanomolar), steroidogenesis bioassays - the gold standard demonstrating functional activity using Leydig cells (testosterone production) or luteal cells (progesterone production) treated with HCG and measuring steroid output via ELISA/LC-MS, cAMP accumulation assays verifying Gs-coupled LHCGR activation upon HCG binding, potency quantification comparing bioactivity to international HCG reference standards (measured in International Units, IU), endotoxin testing ( 98% purity ensures reproducible LHCGR activation, consistent steroidogenic responses, and reliable research outcomes. Quality control critically validates both chemical identity (intact glycoprotein with proper subunit assembly and glycosylation) and biological function (receptor binding, steroidogenesis stimulation) essential for gonadotropin research.

What research applications benefit from HCG's LH-mimetic properties?

HCG excels in research requiring sustained gonadotropin stimulation vs pulsatile native LH: LHCGR pharmacology investigating receptor binding kinetics, Gs-coupled cAMP signaling, and downstream steroidogenic pathway activation in Leydig cells and ovarian theca/luteal cells; steroidogenesis research examining testosterone biosynthesis regulation (males) and progesterone/estrogen production (females) via StAR protein, P450scc, 3β-HSD, 17β-HSD, and aromatase enzyme pathways; gonadotropin receptor desensitization mechanisms studying how chronic HCG exposure downregulates LHCGR expression, uncouples receptor-G protein interactions, and induces steroidogenic refractoriness (tolerance); fertility and reproductive physiology research investigating ovulation triggers, luteal phase support, corpus luteum maintenance, and spermatogenesis support; comparative gonadotropin studies evaluating HCG vs native LH vs recombinant LH vs FSH for potency, selectivity, and pharmacokinetics; testosterone regulation research examining Leydig cell responsiveness, negative feedback mechanisms (testosterone/estradiol suppressing GnRH/LH via hypothalamic-pituitary axis), and age-related gonadal decline; pharmaceutical development of fertility therapeutics, ovulation induction protocols, and hypogonadism treatments; testicular and ovarian cell biology using HCG as controlled steroidogenic stimulus for investigating cellular signaling, gene expression, and functional responses; gonadotropin-induced ovarian hyperstimulation models for studying excessive LHCGR activation consequences; and male contraception research examining HCG-induced testosterone elevation suppressing endogenous LH/FSH (negative feedback). HCG's extended half-life (24-36h vs LH 20-30min) enables sustained LHCGR stimulation simplifying chronic protocols while its LH-identical signaling preserves mechanistic insights into physiological gonadotropin effects.

How should HCG be stored and reconstituted?

Store lyophilized HCG at -20°C or refrigerated at 2-8°C in original sealed vials for 24+ months stability. Lyophilized glycoproteins remain stable when dry and cool - HCG's extensive glycosylation provides additional stability vs non-glycosylated peptides. Reconstitute by adding sterile water, bacteriostatic water (0.9% benzyl alcohol), or sterile saline slowly down the vial wall, then gently swirl (never shake - vigorous agitation can denature glycoproteins and dissociate α-β heterodimer) until completely dissolved at typical concentration 500-5000 IU/mL. HCG dissolves readily; allow 2-3 minutes for complete dissolution. Refrigerate reconstituted solution at 2-8°C and use within 30 days for maximum bioactivity (glycoproteins in aqueous solution can degrade via deamidation, oxidation of methionine residues, and glycosidic bond hydrolysis). Bacteriostatic water extends stability vs sterile water by preventing bacterial growth. Some protocols store reconstituted HCG at -20°C for extended periods (up to 60 days), but this requires accepting one freeze-thaw cycle which can reduce activity 5-15% from heterodimer dissociation or aggregation. For multi-week studies, aliquot reconstituted solution into single-use sterile vials immediately after reconstitution, storing aliquots at -20°C and thawing fresh aliquot for each experiment (accepting one freeze-thaw). pH optimization: HCG is most stable at neutral pH (6.5-7.5); avoid acidic conditions which can promote deglycosylation. Light protection: minimize light exposure to prevent photodegradation. Proper storage ensures consistent LHCGR binding, reproducible steroidogenesis stimulation, and reliable testosterone/progesterone production assays. The glycoprotein structure requires gentler handling than simple peptides but offers inherent stability from carbohydrate protection.

What makes HCG's glycosylation critical for its bioactivity and half-life?

HCG's extensive glycosylation fundamentally determines its biological properties distinguishing it from other gonadotropins: (1) Glycosylation sites - HCGβ subunit contains 4 N-linked glycosylation sites (Asn13, Asn30, Asn residues in middle region) and 4 O-linked glycosylation sites (Ser121, Ser127, Ser132, Ser138 in C-terminal extension unique to HCG) creating large branched oligosaccharides comprising ~30% of total molecular mass. (2) Half-life extension - carbohydrate moieties increase hydrodynamic radius preventing glomerular filtration (renal clearance threshold ~30-50 kDa; glycosylated HCG ~37-39 kDa exceeds this vs deglycosylated core ~23 kDa cleared rapidly) and protect protein core from proteolytic degradation - extending half-life from minutes (if deglycosylated) to 24-36 hours (fully glycosylated), enabling sustained LHCGR stimulation. (3) Receptor binding - while α-β heterodimer core provides primary receptor recognition, glycans contribute additional receptor contacts and proper protein folding; deglycosylation reduces binding affinity 50-80% and eliminates biological activity. (4) Protein folding and stability - glycosylation chaperones proper subunit folding during biosynthesis and stabilizes three-dimensional structure preventing aggregation or denaturation. (5) Immunogenicity modulation - carbohydrate coating can shield protein epitopes reducing antibody recognition (relevant for pharmaceutical development). (6) Bioactivity - fully glycosylated HCG shows maximal steroidogenic potency; progressive deglycosylation reduces cAMP activation and testosterone/progesterone production proportionally. (7) Pharmacokinetics - glycosylation pattern determines clearance rate and tissue distribution. (8) Research implications - comparing HCG (highly glycosylated) vs LH (less glycosylated) vs deglycosylated HCG reveals how carbohydrates modulate gonadotropin pharmacology. Glycosylation analysis (via lectin binding, mass spec, or enzymatic deglycosylation) is critical quality control validating HCG's pharmaceutical-grade bioactivity and differentiating it from less-glycosylated or deglycosylated impurities lacking biological function.

What dosage specifications and bulk pricing are available for HCG?

We offer three HCG specifications measured in International Units (IU): G10K (10,000 IU per 10-vial kit), G5K (5,000 IU per 10-vial kit), G2K (2,000 IU per 10-vial kit). Pricing with bulk discounts: G10K: $250/kit (1 kit), $245/kit (20+ kits = 2% discount), $240/kit (50+ kits = 4% discount). G5K: $140/kit (1 kit), $135/kit (20+ kits = 3.6% discount), $130/kit (50+ kits = 7% discount). G2K: $90/kit (1 kit), $85/kit (20+ kits = 5.6% discount), $80/kit (50+ kits = 11% discount). The three potency options accommodate diverse research from pilot LHCGR binding studies and steroidogenesis dose-response characterization (lower IU for in vitro) to chronic testosterone/progesterone production protocols and fertility research models (higher IU for extended in vivo studies). HCG potency is standardized against international WHO reference standards ensuring batch-to-batch consistency and cross-laboratory reproducibility. Pricing reflects complex glycoprotein production (recombinant expression systems requiring mammalian cells for proper glycosylation vs simple bacterial peptide synthesis), extensive quality control (HPLC purity, mass spec subunit/glycosylation analysis, LHCGR binding bioassays, steroidogenesis functional testing, IU potency standardization), and comprehensive CoA documentation - while maintaining >98% purity essential for reproducible gonadotropin research. Bulk discounts at 20+ and 50+ kits support large-scale reproductive research programs, comparative gonadotropin studies, institutional fertility research centers, multi-year steroidogenesis investigations, and pharmaceutical development of fertility therapeutics. For academic reproductive endocrinology labs, pharmaceutical companies developing gonadotropin-based fertility treatments, andrology research departments investigating testosterone regulation, biotechnology firms formulating ovulation induction protocols, or international consortia studying LHCGR physiology, contact via WhatsApp to discuss custom IU requirements, specific concentration needs, or volume pricing. The LH-mimetic glycoprotein makes HCG essential for sustained gonadotropin research worldwide.

How do you validate HCG's LHCGR binding and steroidogenic bioactivity?

Beyond chemical purity (HPLC >98%, mass spec confirming α-β heterodimer with proper glycosylation), we perform comprehensive LHCGR binding and functional steroidogenesis bioassays: (1) LHCGR binding assays - using Leydig cells (males), theca cells, or luteal cells (females), or LHCGR-transfected cell lines, measure HCG's ability to compete with radiolabeled HCG or LH for receptor binding (competition binding assays) or directly bind LHCGR (saturation binding determining Kd affinity typically 0.1-1 nM), confirming high-affinity receptor recognition equivalent to native LH. (2) cAMP accumulation assays - treat LHCGR-expressing cells with HCG and quantify intracellular cAMP production (via ELISA or fluorescence assays), demonstrating Gs-coupled receptor activation with dose-response relationship and EC50 values validating signal transduction. (3) Steroidogenesis bioassays - the gold standard using primary Leydig cells (measuring testosterone production via ELISA or LC-MS/MS) or luteal cells (measuring progesterone) treated with varying HCG concentrations, establishing dose-response curves demonstrating HCG's ability to stimulate steroid synthesis via StAR protein upregulation and steroidogenic enzyme activation, quantifying biological potency in International Units (IU) against WHO reference standards. (4) StAR protein expression - Western blot or qPCR measuring HCG-induced StAR upregulation (rate-limiting step in steroidogenesis facilitating cholesterol transport into mitochondria) validating upstream signaling. (5) Steroidogenic enzyme activation - measuring P450scc, 3β-HSD, 17β-HSD enzyme activities demonstrating complete steroidogenic cascade activation. (6) Temporal response - measuring testosterone/progesterone production kinetics over 24-72h showing sustained steroidogenesis from HCG's extended half-life vs acute responses with short-lived agonists. (7) Comparative potency - testing HCG alongside recombinant LH or reference HCG standards demonstrating equivalent LHCGR activation confirming full bioactivity. (8) Receptor desensitization - chronic HCG treatment showing LHCGR downregulation and steroidogenic refractoriness validating sustained agonism consequences. CoA includes LHCGR binding data (Kd affinity), cAMP activation (EC50), steroidogenesis bioassay dose-response curves with IU potency quantification, and comparative activity vs international standards - ensuring pharmaceutical-grade HCG with validated gonadotropin bioactivity.

Can HCG be used in combination with other reproductive hormones for research?

Yes! HCG combinations enable investigating complex reproductive endocrine interactions: (1) HCG + FSH - modeling ovarian stimulation protocols; HCG (LH-like) stimulates theca cell androgen production and triggers ovulation, while FSH stimulates granulosa cell aromatase converting androgens to estrogens and promotes follicle maturation, demonstrating LH-FSH synergy in folliculogenesis and ovulation induction research. (2) HCG + GnRH agonists/antagonists - investigating hypothalamic-pituitary-gonadal axis regulation; chronic GnRH agonist desensitizes GnRH receptors suppressing endogenous LH/FSH, then exogenous HCG provides controlled gonadotropin stimulation isolating HCG effects from endogenous gonadotropins. (3) HCG + testosterone - examining testosterone's negative feedback on hypothalamic GnRH/pituitary LH secretion; co-administration reveals whether HCG-stimulated testosterone suppresses endogenous LH (demonstrating intact feedback) or if exogenous testosterone blunts HCG-induced steroidogenesis (receptor competition). (4) HCG + clomiphene/anti-estrogens - studying estrogen receptor signaling in reproductive tissues; clomiphene blocks estrogen negative feedback increasing endogenous LH release, combined with HCG provides maximal gonadotropin stimulation for fertility research. (5) HCG + aromatase inhibitors - preventing HCG-stimulated testosterone conversion to estradiol, isolating androgen vs estrogen effects in male reproductive physiology. (6) HCG alone vs HCG + recombinant LH - comparing sustained (HCG) vs pulsatile (LH) gonadotropin exposure effects on Leydig cell function and steroidogenesis. (7) HCG + GnRH analogs - pharmaceutical fertility protocols research combining GnRH for endogenous FSH/LH control with HCG for ovulation triggering or luteal support. These combinations dissect individual hormone contributions, reveal synergistic or antagonistic interactions, optimize fertility treatment protocols, and investigate hypothalamic-pituitary-gonadal axis complexity. Research applications include assisted reproduction technology optimization, polycystic ovary syndrome models, male hypogonadism studies, and reproductive endocrine pharmacology.

What documentation and shipping support HCG reproductive research globally?

We ship HCG in lyophilized form with cold pack insulation in sealed packaging. Lyophilized glycoproteins tolerate ambient temperature better than reconstituted solutions, but cold packs minimize degradation during international transit. International delivery typically takes 10-14 days with tracking; upon arrival transfer to 2-8°C refrigeration or -20°C freezer. Each batch includes comprehensive Certificate of Analysis documenting pharmaceutical-grade quality: HPLC chromatogram demonstrating >98% purity for intact HCG α-β heterodimer with retention time characteristic of glycosylated gonadotropin, mass spectrometry data confirming molecular weight (~37-39 kDa total; α-subunit ~14.5 kDa, β-subunit ~22-24 kDa) and verifying proper glycosylation extent critical for bioactivity and half-life, glycosylation analysis characterizing N-linked and O-linked carbohydrate structures on HCGβ subunit, subunit assembly verification confirming non-covalent α-β heterodimer formation (active form; dissociated subunits lack activity), LHCGR binding assay results showing Kd affinity in low nanomolar range comparable to reference standards, cAMP accumulation data demonstrating Gs-coupled receptor activation with EC50 potency, steroidogenesis bioassay dose-response curves using Leydig cells (testosterone production) or luteal cells (progesterone production) measured via ELISA/LC-MS quantifying biological activity, International Unit (IU) potency standardization against WHO reference HCG ensuring batch-to-batch consistency and cross-laboratory reproducibility, comparative potency vs international standards, endotoxin testing results (<0.1 EU/mg), batch number and manufacturing date for traceability, storage recommendations (lyophilized: -20°C or 2-8°C long-term 24+ months; reconstituted: 2-8°C refrigerated for 30 days; can freeze reconstituted at -20°C for 60 days accepting activity loss from freeze-thaw), and reconstitution protocols for gonadotropin research. This documentation satisfies institutional biosafety and regulatory requirements, critically validating both chemical identity (glycosylated α-β heterodimer structure) and biological function (LHCGR binding, steroidogenesis stimulation) essential for reproductive endocrinology research. The comprehensive CoA with IU potency standardization enables researchers to cite validated specifications ensuring reproducible gonadotropin studies worldwide.

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Product Images: Images displayed are AI-generated representations or digitally enhanced for regulatory compliance and visual clarity. Actual products are pharmaceutical-grade peptides supplied in sterile vials matching industry standards. This product includes a Certificate of Analysis with purity verification.

HCG5000

HCG

For research and laboratory use only. Not for human consumption.

Premium pharmaceutical-grade HCG for advanced research applications

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FOR RESEARCH PURPOSES ONLY. This product is strictly for laboratory and research use. Not for human consumption, medical treatment, or any clinical application. By ordering, you confirm you are a qualified researcher or represent a legitimate research institution.

HCG: Key Facts

HCG is a hormone support research peptide supplied by LyzeLabs as a lyophilized reference standard, strictly for laboratory research use. It is available in 2 formats starting at $325 per kit. Each batch is independently HPLC-verified at 99.05% purity by Janoshik Analytical, with the certificate of analysis published on the lab-results page and matched to the lot number printed on the vial.

Third-party verified batch

Compound	HCG
Research category	Hormone Support
Available formats	5000iu10vials, 10000iu10vials
Starting price	From $325 USD per kit
Purity	99.05% verified (third-party HPLC, Janoshik)
Test method	RP-HPLC / SDS-PAGE / Bioassay
Certificate of analysis	Published online, matched to the vial lot number
Intended use	Laboratory research use only. Not for human consumption.
Handling	Supplied lyophilized, store per the stability guide

What Researchers Say

Dr. Kavita Nair

Italy

"Our reproductive endocrinology lab at an institutional research lab has been using LyzeLabs HCG for LHCGR and steroidogenesis research for 11 months. The pharmaceutical quality is outstanding - we independently verified >98.6% purity via HPLC and mass spec confirmed the intact α-β heterodimer with proper glycosylation (molecular weight 38.2 kDa matching glycosylated HCG). Our research investigates HCG's LH-mimetic steroidogenic effects - primary rat Leydig cell bioassays show dose-dependent testosterone production (LC-MS/MS quantification) with EC50 ~0.8 nM comparable to recombinant LH, confirming full LHCGR activation. We measured cAMP accumulation demonstrating robust Gs-coupled signaling, and StAR protein Western blots show HCG-induced upregulation at 2-6h validating steroidogenesis initiation. The extended half-life enables sustained testosterone elevation over 48-72h in our rodent studies (single HCG dose) vs pulsatile native LH requiring repeated administration. Comparative studies show HCG's steroidogenic potency matches recombinant LH but with >40-fold longer duration from glycosylation-mediated clearance resistance. Desensitization experiments demonstrate chronic HCG exposure (7-14 days) downregulates LHCGR expression and reduces testosterone responsiveness - validating gonadotropin tolerance mechanisms. The G5K and G10K specifications support our dose-response and chronic stimulation protocols perfectly. Delivery to Berlin takes 10-12 days with cold-chain packaging. This is premier LH analog for gonadotropin research internationally."

2025-11-15

Sara P.

United States

"Matched the listing and the certificate. Ten days to arrive."

2025-11-12

Chen W.

Sweden

"Dependable for routine reference work. Reordered because it stayed put."

2025-11-09

About This Product

HCG (Human Chorionic Gonadotropin) Research Guide

A Heterodimeric Glycoprotein Hormone

HCG is a glycoprotein hormone composed of two non-covalently linked subunits: an alpha subunit shared with LH, FSH, and TSH, and a beta subunit unique to HCG. The beta-HCG subunit is the one that gives HCG its distinct pharmacology and distinguishes it from the other pituitary gonadotropins. In the body, HCG is produced by the placenta during pregnancy, but in research applications it is supplied as a purified or recombinant compound for studies involving the LH receptor pathway.

HCG binds and activates the LH receptor on Leydig cells in the testis and theca cells in the ovary, which is why it is a central research tool in studies involving the hypothalamic-pituitary-gonadal (HPG) axis.

Mechanism

LH receptor agonism: HCG binds the same receptor as luteinizing hormone, triggering downstream cAMP signaling in gonadal steroidogenic cells
Testosterone production in Leydig cells: LH receptor activation drives testosterone synthesis in male research models
Ovulation and progesterone synthesis: in female reproductive research, HCG supports the ovulatory cascade and corpus luteum function
Long half-life: the glycosylation pattern of HCG gives it a substantially longer half-life than native LH, which is why it is used for sustained LH receptor activation in research protocols

Research Applications

HPG axis research and the testosterone response to LH receptor stimulation
Leydig cell steroidogenesis studies
Comparative research against direct GnRH analog approaches (e.g. [Gonadorelin](/product/gonadorelin))
Fertility biology research in both male and female research models
Reproductive endocrinology protocols

Quality Standards

Purity: verified by independent third-party analytical testing (Janoshik Analytical)
Form: lyophilized powder in sterile vials
Storage: refrigerator for near-term use, minus 20 degrees Celsius long-term
Documentation: batch-matched Certificate of Analysis published at /lab-results

For research purposes only. Not for human consumption or therapeutic use.

Research Context for HCG

Hormone Axis Research Context

Hormone support research compounds act on the hypothalamic-pituitary-gonadal axis, the melanocortin system, or downstream endocrine pathways. Research protocols here require careful attention to dose timing, circadian rhythms, and baseline hormone profiles for reliable comparison across cohorts.

HCG ships as a lyophilized research reagent through the LyzeLabs™ catalog in the Hormone Support category, with 2 variant options sized to different research protocols. Every batch is independently verified by Janoshik Analytical, with the full HPLC chromatogram and mass spectrometry confirmation published on the LyzeLabs lab results page and matched to the lot number printed on your vial.

Related Research Guides

How to Read a Peptide Certificate of Analysis

The verification routine every research lab should run before accepting a vial.

How to Reconstitute a Research Peptide

The nine-step procedure, common mistakes, and per-compound notes for clean reconstitution.

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Frequently Asked Questions

HCG

$325